Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
4182636 | L'Encéphale | 2008 | 7 Pages |
Abstract
The deficit of inhibition of return seems to be time-stable and to be unrelated to psychopathology or length of illness. The contribution of neuroleptic medication to this deficit cannot be determined. Recent data suggest a deficit of inhibition of return in two human models of psychosis (dimethyltryptamine and ketamine). Further studies should clarify whether blunted inhibition of return might represent a trait marker of schizophrenia.
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Authors
O. Kebir, O. Ben Azouz, I. Amado, K. Tabbane,