| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4271377 | The Journal of Sexual Medicine | 2010 | 10 Pages |
Abstract
Activated NAD(P)H oxidase in the penis is an initial source of oxidative stress resulting in eNOS uncoupling, thus providing a mechanism of eNOS uncoupling and endothelial dysfunction in hypercholesterolemia-induced ED. Musicki B, Liu T, Lagoda GA, Strong TD, Sezen SF, Johnson JM, and Burnett AL. Hypercholesterolemia-induced erectile dysfunction: Endothelial nitric oxide synthase (eNOS) uncoupling in the mouse penis by NAD(P)H oxidase.
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Authors
Biljana PhD, Tongyun MS, Gwen A. MS, Travis D. BA, Sena F. PhD, Justin M. BA, Arthur L. MD,