| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4271505 | The Journal of Sexual Medicine | 2011 | 8 Pages |
Abstract
These findings provide a molecular basis for chronically reduced eNOS function in the penis by SCD, which involves decreased eNOS phosphorylation on Serâ1177 and decreased eNOSâHSP90 interaction. Musicki B, Champion HC, Hsu LL, Bivalacqua TJ, and Burnett AL. Postâtranslational inactivation of endothelial nitric oxide synthase in the transgenic sickle cell mouse penis. J Sex Med 2011;8:419-426.
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Authors
Biljana PhD, Hunter C. MD, PhD, Lewis L. MD, PhD, Trinity J. MD, PhD, Arthur L. MD,