| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4322860 | Neuron | 2007 | 4 Pages |
Abstract
The cellular energy-sensing kinase AMPK is known to be activated in neurons in response to metabolic insults, but the downstream consequences have been unclear. A study by Kuramoto and colleagues in this issue of Neuron favors the idea that AMPK activation is neuroprotective, and suggests a potential mechanism for this effect involving phosphorylation of the GABAB receptor.
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Authors
D. Grahame Hardie, Bruno G. Frenguelli,