| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4326430 | Brain Research | 2010 | 13 Pages |
Abstract
âºForebrain ischemia induced the neuronal apoptosis. âºCaspase-3 and Bcl-2 mRNA were increased at 24 h after forebrain ischemia. âºForebrain ischemia induced the increase of caspase-3 and Bcl-2 protein expression. âºApoptotic neurons were decreased after propofol treatment. âºCaspase-3 mRNA and protein expression were inhibited after propofol treatment. âºBcl-2 mRNA and protein expression were increased after propofol treatment.
Keywords
N-methyl-d-aspartatePaO2B-cell leukemia-2PaCO2APAf-1MCAONMDABcl-2FASRT–PCRapoptotic protease activating factor-1γ-aminobutyric acidmiddle cerebral artery occlusionForebrain ischemiaTerminal deoxynucleotidyl transferase-mediated dUTP nick end labelingTUNELApoptosisReperfusionarterial oxygen pressuremean arterial blood pressurearterial carbon dioxide pressuremapreverse transcription–polymerase chain reactionPropofolCaspase-3GABA
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Authors
Jun Li, Baoqing Han, Xuesong Ma, Sihua Qi,