| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4327870 | Brain Research | 2009 | 11 Pages |
Abstract
It may be possible to protect catecholaminergic neurons from reactive oxygen species-induced apoptotic death by not only blocking catecholamine synthesis, but also, by inhibiting carbonic anhydrase-dependent chloride/bicarbonate exchange with acetazolamide or chlorthalidone. These agents may prove salutary in reducing cell death in patients with traumatic brain injury or stroke.
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Authors
Ryan Woodman, Warren Lockette,