| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 4338285 | Neuroscience | 2012 | 14 Pages |
Abstract
⺠Postsynaptic depolarisation accelerates the hypoxic depression of glutamate release. ⺠This process is Ca2+- and PKC-dependent and may entail postsynaptic membrane fusion. ⺠In addition, this process requires activation of GI mGluRs, but not CB1 receptors. ⺠We propose a novel form of retrograde signalling activated by metabolic stress.
Keywords
PKC(RS)-1-aminoindan-1,5-dicarboxylic acidQX-314t50N-(piperidin-1-yl)-5-(4-iodophenyl)-1-(2,4-dichlorophenyl)-4-methyl-1H-pyrazole-3-carboxamideGroup I metabotropic glutamate receptorAM251AIDAfEPSPN-ethylmaleimideDPCPXNSF8-Cyclopentyl-1,3-dipropylxanthineEPSCVsplong-term potentiationLTPexcitatory postsynaptic currentN-ethylmaleimide-sensitive factorProtein kinase C inhibitorNEMField excitatory postsynaptic potentialProtein kinase C
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Authors
F. Nuritova, B.G. Frenguelli,