Article ID Journal Published Year Pages File Type
4342604 Neuroscience 2006 6 Pages PDF
Abstract

Hyperammonemia is responsible for many of the neurological alterations in patients with hepatic encephalopathy by mechanisms that remain unclear. Hyperammonemia alters phosphorylation of brain protein kinase C substrates and impairs N-methyl-d

Related Topics
Life Sciences Neuroscience Neuroscience (General)
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