Article ID Journal Published Year Pages File Type
4345412 Neuroscience Letters 2011 5 Pages PDF
Abstract
► Successful prophylaxis will require a focus on more than one pathophysiological process. ► Choice of experimental model depends on the specific experimental issue to be addressed. ► Disinhibition and new excitatory synapse formation are key prophylactic targets. ► Enhancing trophic support by BDNF may “rescue” malfunctioning injured interneurons. ► Blockade of thrombospondin actions at a 2δ-1 receptors after injury is antiepileptogenic.
Related Topics
Life Sciences Neuroscience Neuroscience (General)
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