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Nitric oxide in glutamate-induced compound action potential threshold shifts

Article ID Journal Published Year Pages File Type
4356088 Hearing Research 2008 6 Pages PDF
Abstract
NO mediates excitotoxicity when the cochlea is perfused with l-glutamate.
Keywords
7-NIEAAcGMPnNOSNOSN-methyl-d-aspartatei.p.NMDAAMPA7-nitroindazoleAPscAMPl-NAMEl-NG-Nitroarginine methyl esterONOO−Cyclic adenosine monophosphateexcitatory amino acidexcitatory amino acidsexcitotoxicityCochleaintraperitonealCNSneuronal nitric oxide synthasecentral nervous systemmean arterial blood pressurecyclic guanosine monophosphatemapNitric oxidenitric oxide synthaseCompound Action PotentialCaPCa+2glutamateCalcium ions
Related Topics
Life Sciences Neuroscience Sensory Systems
Preview
Nitric oxide in glutamate-induced compound action potential threshold shifts
Authors
Mihir R. Patel, Jocelyn C. Stamat, Carlton J. Zdanski, Charles S. Jr., Jiri Prazma,
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Journal
Hearing Research
Journal: Hearing Research
Related Categories
7-NI
EAA
cGMP
nNOS
NOS
N-methyl-d-aspartate
i.p.
NMDA
AMPA
7-nitroindazole
APs
cAMP
l-NAME
l-NG-Nitroarginine methyl ester
ONOO−
Cyclic adenosine monophosphate
excitatory amino acid
excitatory amino acids
excitotoxicity
Cochlea
intraperitoneal
CNS
neuronal nitric oxide synthase
central nervous system
mean arterial blood pressure
cyclic guanosine monophosphate
map
Nitric oxide
nitric oxide synthase
Compound Action Potential
CaP
Ca+2
glutamate
Calcium ions
Behavioral Neuroscience
Biological Psychiatry
Cellular and Molecular Neuroscience
Cognitive Neuroscience
Developmental Neuroscience
Endocrine and Autonomic Systems
Neurology
Neuroscience (General)
Sensory Systems
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