| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5500946 | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease | 2017 | 43 Pages |
Abstract
The present study provides compelling evidences that FP combined with FO may contribute to revert some processes related to steroid resistance induced by oxidative stress due to cigarette smoke exposure increasing the anti-inflammatory effects of FP.
Keywords
CSELong-acting beta2-agonistsHDAC3HDAC2ICSERKSDANF-κBextracellular signal-regulated kinase1/2Cigarette smokeBronchial epithelial cellscigarette smoke extractsnuclear factor-κBFluticasone propionateHistone deacetylase 2histone deacetylase 3CorticosteroidsInhaled corticosteroidsglucocorticoid receptor
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Authors
M. Ferraro, M. Gjomarkaj, L. Siena, S. Di Vincenzo, E. Pace,