| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5501041 | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease | 2017 | 41 Pages |
Abstract
Atherosclerosis-prone conditions downregulated endothelial Nox4 to accelerate the progress of atherosclerosis, at least in part, by upregulating sEH to enhance inflammation.
Keywords
EETsFBSVCAM1SMCPWVLSSEDNSTZeNOSNOX4OROEndothelial Basal Medium-2EBM-2ROSsEHsiRNAAtherosclerosisLCALaminar shear stressOscillatory shear stressstreptozotocinepoxyeicosatrienoic acidsEndotheliumOSSHAECsmall interference RNAType I diabetesOil red OWestern dietnormal dietPulse wave velocityfetal bovine serumSmooth muscle cellsEndothelial cellshuman aortic endothelial cellsleft common carotid arteryendothelial nitric oxide synthaseVascular cell adhesion molecule 1Nitric oxideReactive oxygen species
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Authors
Pingping Hu, Xiaojuan Wu, Alok R. Khandelwal, Weimin Yu, Zaicheng Xu, Lili Chen, Jian Yang, Robert M. Weisbrod, Kin Sing Stephen Lee, Francesca Seta, Bruce D. Hammock, Richard A. Cohen, Chunyu Zeng, Xiaoyong Tong,