Intermittent hypoxia confers pro-metastatic gene expression selectively through NF-ÎºB in inflammatory breast cancer cells

Article ID	Journal	Published Year	Pages	File Type
5501844	Free Radical Biology and Medicine	2016	48 Pages	PDF

Keywords

BSP NAC HO-1 COX-2 SFN IL-1β N-acetyl-l-cysteine ADAM19 IL1A AP-1 ECM CIH TFBS tert-butylhydroquinone HIF IκBα MMP2 MMP9 CA9 SMC1 Jnk Nrf2 DMF XRCC2 TBHQ IBC NF-κB TNC Dcf TREM1 keap1 c-Jun N-terminal kinase MAPK Bisulfite sequencing PCR ROS tenascin-C Interleukin-1α Interleukin-1β Oxidative stress dimethyl fumarate transcription factor binding site Inflammatory breast cancer Sulforaphane Cyclooxygenase-2 NF-E2-related factor 2 antioxidant response element Hypoxia-inducible factor nuclear factor κB Extracellular matrix matrix metalloproteinase 2 Matrix metalloproteinase 9 ARE heme oxygenase 1 intermittent hypoxia chronic intermittent hypoxia activator protein-1 Kelch-like ECH-associated protein 1 mitogen-activated protein kinase Carbonic anhydrase IX Reactive oxygen species Estrogen receptor triggering receptor expressed on myeloid cells

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

Intermittent hypoxia confers pro-metastatic gene expression selectively through NF-ÎºB in inflammatory breast cancer cells

Authors

Katrin Gutsche, Elisa B. Randi, Volker Blank, Daniel Fink, Roland H. Wenger, Cornelia Leo, Carsten C. Scholz,