Zinc regulates Nox1 expression through a NF-ÎºB and mitochondrial ROS dependent mechanism to induce senescence of vascular smooth muscle cells

Article ID	Journal	Published Year	Pages	File Type
5502036	Free Radical Biology and Medicine	2017	28 Pages	PDF

Keywords

PIC fluorescein-di-β-D-galactopyranoside FluoZin-3 VSMC SA-β-gal ACD CHX FDG TPEN Chl NAC DPBS MEF FBS PBS DMEM EGF NF-κB 2′,7′-dichlorodihydrofluorescein diacetate Dulbecco's modified Eagle Medium H2DCFDA NOx ROS Angiotensin II Iron actinomycin D NADPH oxidase Cardiovascular diseases TERT Telomerase Ang II CVD SOD fetal bovine serum Vascular smooth muscle cells Superoxide dismutase cycloheximide epidermal growth factor nuclear factor kappa-B Dulbecco's phosphate-buffered saline Low-density lipoprotein LDL Phosphate-buffered saline Copper Manganese mouse embryonic fibroblasts N-acetyl cysteine Stress-induced premature senescence senescence-associated-β-galactosidase Cobalt Chloroquine protease inhibitor cocktail Reactive oxygen species

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

Zinc regulates Nox1 expression through a NF-ÎºB and mitochondrial ROS dependent mechanism to induce senescence of vascular smooth muscle cells

Authors

G. Salazar, J. Huang, R.G. Feresin, Y. Zhao, K.K. Griendling,