| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5553150 | Biomedicine & Pharmacotherapy | 2017 | 16 Pages |
Abstract
Zinc pyrithione attenuates lethal perfusion-induced injury in a manner that is reliant on ErbB2/PI3K/Akt activity.
Keywords
LVESPKrebs-HenseleitLVDPDAFAARLVEDPRT-PCRkDaGAPDHTTCTSQERBBp38MAPKDEVD-pNAJnkK-HB10BSAc-Jun N-terminal kinaseDMSOERK1/2I/RMTTsiRNAAdenosine TriphosphateATPbovine serum albuminSDS-PAGESodium dodecyl sulfate polyacrylamide gel electrophoresisInfarct sizeIschemiaischemia/reperfusionLeft ventricular developed pressureImmunoprecipitationTriphenyl Tetrazolium ChlorideTUNELcoronary flowdimethyl sulphoxidediaminofluoresceinZinc pyrithioneReperfusionleft ventricular end diastolic pressureZincarea at riskMyocardiumNitric oxideReverse transcriptase-polymerase chain reactionwortmanninp38 mitogen activated protein kinaseCreatine kinaseKilo Daltonglyceraldehyde-3-phosphate dehydrogenase
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Authors
Sandhya Thokala, Santhipriya Inapurapu, Vijaya Lakshmi Bodiga, Praveen Kumar Vemuri, Sreedhar Bodiga,