Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5557024 | Life Sciences | 2017 | 29 Pages |
Abstract
Taken together, our findings indicated that Cd exposition induced dose-dependent testicular dystrophic calcification, which was associated with increased antioxidant enzymes activity and protein oxidation, mineral imbalance, germ cell calcification and death in rats. Although increased CAT and SOD activity represents a counter-regulatory reaction to cadmium-induced toxicity, this reaction is insufficient to prevent testicular pathological remodeling.
Keywords
IP3EDSCaMKIIInositol trisphosphateCdCl2PKCCATGSTAntioxidant enzymesIronEnergy Dispersive X-ray SpectroscopySeminiferous epitheliumvolume densitydiacylglycerolDAGUltrastructureCAMSODSeleniumSuperoxide dismutaseZincSERCASeminiferous tubulesCopperTesticular toxicityMagnesiumElectron microscopyProtein kinase CCatalaseCadmiumCalmodulinCadmium chlorideCalciumcalcium/calmodulin-dependent kinase IIglutathione S-transferase
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Authors
Marli do Carmo Cupertino, Rômulo Dias Novaes, Eliziária Cardoso Santos, Daniel Silva Sena Bastos, Daiane Cristina Marques dos Santos, Maria do Carmo Queiroz Fialho, Sérgio Luis Pinto da Matta,