| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5561801 | Toxicology | 2017 | 39 Pages |
Abstract
In conclusion, AChE activity and apoptosis were induced in SH-SY5Y cells and mice treated with ethanol, which may indicate that increased AChE may related to apoptosis induced by ethanol. Unusually high AChE activity may be an effect marker of exposure to ethanol. The relationship between AChE and apoptosis might represent a novel mechanism of ethanol-associated neuronal injury.
Keywords
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Authors
Wenjun Sun, Liangjing Chen, Wei Zheng, Xiaoan Wei, Wenqi Wu, Ellen G. Duysen, Wei Jiang,