| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5585213 | Bone | 2017 | 10 Pages |
Abstract
These data suggests that ACKR2 functions as a regulator of mechanically-induced bone remodeling by affecting the differentiation and activity of bone cells and the availability of CC chemokines at periodontal microenvironment. Therapeutic strategies based on increase of ACKR2 might be useful to hinder bone loss in inflammatory conditions.
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Authors
Izabella Lucas de Abreu Lima, Janine Mayra da Silva, LetÃcia Fernanda Duffles Rodrigues, Davidson Frois Madureira, Angélica Cristina Fonseca, Gustavo Pompermaier Garlet, Mauro Martins Teixeira, Remo Castro Russo, Sandra Yasuyo Fukada,