| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5586138 | Placenta | 2017 | 8 Pages |
Abstract
This study provides evidence that dysregulated IKCa and SKCa channels might contribute to fetal pathogenesis of PE through direct promotion of vascular constriction of CPAs and through affecting functions of NO and activities of NOS.
Keywords
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Biochemistry, Genetics and Molecular Biology
Developmental Biology
Authors
Fan-fan Li, Meng-zhou He, Yin Xie, Yuan-yuan Wu, Mei-tao Yang, Yao Fan, Fu-yuan Qiao, Dong-rui Deng,