Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5586177 | Placenta | 2017 | 9 Pages |
Abstract
The lack of pSmad2C in villous CTBs suggests that other mitogens than TGF-β could promote Smad2 linker phosphorylation under homeostatic conditions. Whereas autocrine signalling activates Smad2/3 in differentiating EVTs, paracrine factors contribute to Smad phosphorylation in these cells.
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Authors
S. Haider, V. Kunihs, C. Fiala, J. Pollheimer, M. Knöfler,