| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5588464 | Metabolism | 2017 | 12 Pages |
Abstract
Our findings support the notion that caloric restriction represents an important mechanism to explain the very early anti-diabetic effects observed after bariatric surgery. However, exclusion of the upper gastrointestinal tract also provides further metabolic improvements, possibly mediated by gastrointestinal hormonal responses and altered postprandial glucose absorption.
Keywords
RYGBHbA1cFPGSGT1PYYGIPT2DGLP-1HOMA-IRBMRISRAUCISIBody weight lossBiliopancreatic diversion with duodenal switchβ-Cell functionEnzyme-linked immunosorbent assayELISAtumor necrosis factor alphaInsulin sensitivityType 2 diabetesbody mass indexBMIDisposition indexTNF-αFasting plasma glucosearea under the curveInsulin secretion ratebasal metabolic rateHemoglobin A1cPancreatic polypeptideglucose-dependent insulinotropic peptideSleeve gastrectomyglucagon like peptide-1
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Authors
Andréanne Michaud, Thomas Grenier-Larouche, Dominique Caron-Dorval, Simon Marceau, Laurent Biertho, Serge Simard, Denis Richard, André Tchernof, André C. Carpentier,