| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5589576 | Gene | 2017 | 28 Pages |
Abstract
LncRNA-ATB/miR-200a/β-catenin regulatory axis likely contributed to the development of liver fibrosis in HCV patients. Knockdown of lncRNA-ATB might be a novel therapeutic target for HCV-related liver fibrosis.
Keywords
ECMTGF-βCOL1A1HSCslncRNAssiRNAsα-SMA3′-UTR3′-untranslated regionHCCLong non-coding RNAssmall interfering RNAsα-smooth muscle actinβ-catenintransforming growth factor-betaEMTconditioned mediaCompeting endogenous RNAHepatic stellate cellsceRNALong noncoding RNALiver fibrosisExtracellular matrixHCVHepatitis C virusHepatocellular carcinomaEpithelial-mesenchymal transition
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Authors
Fu Na, Zhao Su-Xian, Kong Ling-Bo, Du Jing-Hua, Ren Wei-Guang, Han Fang, Zhang Qing-Shan, Li Wen-Cong, Cui Po, Wang Rong-Qi, Zhang Yu-Guo, Nan Yue-Min,