| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5599269 | Atherosclerosis | 2017 | 43 Pages |
Abstract
Our data suggest that an atherogenic condition could activate eosinophils and modulate the phenotype of macrophages (from M2 to M1 phenotype), in part, through the CD36 receptor signaling.
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Minghui Qin, Lai Wang, Fuqiang Li, Mingjie Yang, Lei Song, Fang Tian, Ada Yukht, Prediman K. Shah, Marc E. Rothenberg, Behrooz G. Sharifi,