| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5599495 | Atherosclerosis | 2017 | 31 Pages |
Abstract
AMPK activation reduces the formation of atheromata-inducing macrophages in ApoEâ/â-deficient mice by inhibiting expression of Ccr2, thereby preventing the Ccr2-mediated migration of Ly6Chi monocytes from the BM. Therefore, AMPK may be a promising target for the treatment of atherosclerosis.
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Authors
Jing Wang, Ang Ma, Ming Zhao, Haibo Zhu,