Article ID Journal Published Year Pages File Type
5602580 Heart, Lung and Circulation 2017 9 Pages PDF
Abstract
These results demonstrated that Exenatide improved cardiac function, increased translocation of GLUTs, and suppressed translocation of FAT/CD36 after myocardial I/R injury. This protective effect was mediated, at least in part, through modulation of the cardiac p38γ MAPK.
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Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Preview
Exenatide Regulates Substrate Preferences Through the p38γ MAPK Pathway After Ischaemia/Reperfusion Injury in a Rat Heart
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