| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5603439 | Heart Rhythm | 2017 | 29 Pages |
Abstract
This study revealed a novel mechanism for IL-1β in contributing to defective excitation-contraction coupling and arrhythmogenesis in the post-MI heart. Our results suggest that inhibition of IL-1 signaling post-MI may represent a novel antiarrhythmic therapy.
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Authors
Nicole M. PhD, Lianguo MD, Johnny BS, Robert R. PhD, Samantha D. MS, Donald M. PhD, Merry L. PhD, Crystal M. PhD, FHRS,