Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5604276 | International Journal of Cardiology | 2017 | 37 Pages |
Abstract
The ability of the kidneys to remove excess intravascular volume is decreased in a substantial amount of euvolemic and optimally treated HFrEF patients. Renal response relates to filtration function and tubular sodium handling. In contrast, loop diuretics can surmount decreased renal tubular sodium excretion but remain dependent on eGFR.
Keywords
ACEHFrEFnatriuresisLVOTRAASNT-proBNPLVEFCVPPRAangiotensin converting enzymeDiuresisrenin-angiotensin systemRenin-angiotensin-aldosterone systemCentral venous pressuremean arterial pressurePlasma renin activityleft ventricular outflow tractheart failureheart failure with reduced ejection fractionSystolic heart failuremapleft ventricular ejection fraction
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Authors
Petra Nijst, Frederik H. Verbrugge, Pieter Martens, Matthias Dupont, W.H. Wilson Tang, Wilfried Mullens,