| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5615098 | Journal of Clinical Lipidology | 2017 | 9 Pages |
Abstract
β3-AR activation increased the activities of the apoA-I promoter, HNF-4, and HNF-3, which might account for the mechanism of β3-AR-mediated upregulation of hepatic apoA-I expression. β3-AR might exert an anti-atherosclerotic effect by upregulating hepatic apoA-I expression and promoting the cholesterol reverse transport process.
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Xiaojing Cao, Yanfang Li,