Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5619798 | Resuscitation | 2017 | 25 Pages |
Abstract
In this study, low CBF after cardiac arrest is not associated with anaerobic metabolism. Hypoperfusion appears to be the consequence of a decrease of neuronal functioning and metabolic needs. Alternatively, hypoperfusion may decrease cerebral metabolism. Subsequently, metabolism increases in survivors, consistent with resumption of neuronal activity, whereas in non-survivors lasting low metabolism reflects irreversible neuronal damage.
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Authors
Cornelia W. Hoedemaekers, Philip N. Ainslie, Stijn Hinssen, Marcel J. Aries, Laurens L. Bisschops, Jeannette Hofmeijer, Johannes G. van der Hoeven,