| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5648919 | Journal of Dermatological Science | 2017 | 9 Pages |
Abstract
These results suggest that fluctuations in chemokine levels may contribute in a coordinated way to the wound-healing process and lend clues toward efficient cell therapies for EB.
Keywords
NHEKHMGB1EBsSLCqRT-PCRBMSCMSCRAGESDF-1RDEBCCL27CTACKMIPFITCDDEBHSCBSAEpidermolysis bullosa simplexELISAEnzyme-linked immunosorbent assayhigh-mobility group box 1JEBstandard error of the meandermo-epidermal junctionDEJbody surface areaMesenchymal stromal cellHematopoietic stem cellBone marrow stem cellsdominant dystrophic epidermolysis bullosastromal cell-derived factor-1fluorescein isothiocyanateSEMbone marrowquantitative reverse transcription polymerase chain reactionmacrophage inflammatory proteinNormal human epidermal keratinocyteChemokinerecessive dystrophic epidermolysis bullosaepidermolysis bullosajunctional epidermolysis bullosaReceptor for advanced glycation end-products
Related Topics
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Authors
Inkin Ujiie, Yasuyuki Fujita, Chihiro Nakayama, Wakana Matsumura, Shotaro Suzuki, Satoru Shinkuma, Toshifumi Nomura, Riichiro Abe, Hiroshi Shimizu,