| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5722228 | Journal of Affective Disorders | 2017 | 11 Pages |
â¢The dissociative PTSD subtype is supported by nearly all included studies.â¢The dissociative subtype can be identified according to the DSM-5 description.â¢Childhood assault emerged as a more consistent risk factor for dissociative PTSD.â¢Research on the impact of dissociative PTSD on prevention and treatment is needed.
BackgroundThe dissociative PTSD (D-PTSD) subtype was first introduced into the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) in 2013. Prior to this, studies using latent profile analysis (LPA) or latent class analysis (LCA), began to provide support for the D-PTSD construct and associated risk factors. This research is important, because dissociative symptoms in the context of PTSD may potentially interfere with treatment course or outcome. The aims of the present study were twofold: to systematically review the LCA and LPA studies investigating support for the D-PTSD construct; and to review the associated research on the risk factors or covariates of D-PTSD in the identified studies.MethodSix databases (PubMed, Web of Science, Scopus, PILOTS, PsychInfo, and Embase) were systematically searched for relevant papers.ResultsEleven studies were included in the present review. The majority of the studies were supportive of the D-PTSD subtype; primarily characterized by depersonalization and derealization. Several covariates of the D-PTSD subtype have been investigated with mixed results.LimitationsMany limitations relate to the state of the current literature, including a small number of studies, the use of self-report measurements of PTSD, and heterogeneity across the samples in investigated covariates.ConclusionThe results were overall supportive of the D-PTSD construct. Future research on D-PTSD and associated risk factors is needed to shed light on the possibilities of facilitating preventive actions, screening, and implications on treatment effects.
