| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5733826 | Journal of Surgical Research | 2017 | 11 Pages |
Abstract
Our results indicate that overexpression of Trx-1 attenuates cardiac dysfunction during CLP. The mechanism of action may involve reduction of oxidative stress, apoptosis, and vascular permeability through activation of Trx-1/HO-1 and anti-apoptotic protein survivin.
Related Topics
Health Sciences
Medicine and Dentistry
Surgery
Authors
Rickesha L. MD, Vaithinathan PhD, Rajesh PhD, Mahesh PhD, Jacob DO, MPH, David W. MD, FACS, Nilanjana PhD, FAHA,