Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5733877 | Journal of Surgical Research | 2017 | 10 Pages |
Abstract
MSP68 downregulates actin cytoskeleton-dependent, Rac1-MAP kinase-mediated neutrophil motility. Thus, MSP68 is a novel therapeutic candidate for regulating inflammation and tissue damage caused by excessive neutrophil migration in sepsis.
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Authors
Louie MD, Monowar PhD, Weng-Lang PhD, Jeffrey MD, Gene F. MD, Marc PhD, Ping MD,