| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5734680 | Surgery | 2017 | 15 Pages |
Abstract
The surplus liver regeneration after ALPPS is probably mediated through parenchymal damage and subsequent release of growth stimulators, which again upregulates genes involved in cellular regeneration and downregulates genes involved in cellular homeostasis. We also demonstrate that growth of the future liver remnant, comparable to that seen after ALPPS, could be achieved by radiofrequency ablation treatment of the deportalized liver, that is, a procedure in which the initial step in humans can be performed percutaneously.
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Authors
Kasper Jarlhelt MD, PhD, Anders Riegels MD, PhD, Betina Norman MD, Michelle MD, Anders Patrik Alexander MSc, Uffe Birk MD, PhD, Jens Randel MD, DMSc, Stephen MD, FRCPath, Frank Viborg MD, DMSc,