| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5832113 | International Immunopharmacology | 2015 | 7 Pages |
Abstract
Taken together, we assume that the transcriptional level of osteoclastogenesis of healthy young humans is not regulated by BDNF, ACh, and nicotine. Thus, these drugs do not seem to worsen bone degradation and might therefore be suitable as modulators of bone substitution materials if having a positive effect on bone formation.
Keywords
b2mM-CSFmAChRTrkBRANKLnAChRTropomyosin-related kinase receptor BTGF-βDMEMPBSPBMCsBDNFBSAcAMPCyclic adenosine monophosphatecycle thresholdbovine serum albuminAChOsteoclastAcetylcholinestandard deviationCarbon dioxideReal-time RT-PCRReal-Time reverse transcriptase polymerase chain reactionperipheral blood mononuclear cellsNon-neuronal cholinergic systemtumor growth factor-βbrain derived neurotrophic factorPhosphate buffered salineMultiple myelomaNicotineChATCarATCarnitine acetyltransferaseCO2choline acetyltransferasemuscarinic acetylcholine receptornicotinic acetylcholine receptor
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Authors
Sebastian Ternes, Katja Trinkaus, Ivonne Bergen, Sven Knaack, Michael Gelinsky, Olaf Kilian, Christian Heiss, Katrin Susanne Lips,