Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
5841437 | Life Sciences | 2016 | 35 Pages |
Abstract
Our findings suggested that PLGF contributed to hyperoxia-induced lung injury through promoting apoptosis and EMT. PLGF may become a therapeutic target of hyperoxia-induced lung injury.
Keywords
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Liang Zhang, Shuang Zhao, Lijie Yuan, Hongmin Wu, Hong Jiang, Gang Luo,