| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5842883 | Life Sciences | 2011 | 8 Pages |
Abstract
1) Concomitantly increased endostatin is a defensive response to the increased VEGF in UC, 2) MMP-9 is a key enzyme to generate endostatin which may modulate the balance between VEGF and endostatin during experimental UC, and 3) endostatin treatment plays a beneficial role in UC. Thus, anti-angiogenesis seems to be a new therapeutic option for UC.
Keywords
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Authors
Ganna Tolstanova, Xiaoming Deng, Tetyana Khomenko, Pallavi Garg, Brankica Paunovic, Longchuan Chen, Shanthi V. Sitaraman, Joseph Shiloach, Sandor Szabo, Zsuzsanna Sandor,