| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5860357 | Toxicology Letters | 2014 | 8 Pages |
Abstract
Overall, our data suggest that the modulation of intracellular redox status induced by C3G may be an important protective mechanism against endothelial damage in hypoxic conditions.
Keywords
HUVECSsulfosalycilic acidNQO-1Heme oxygenase-1C3GOPAHO-1HIF-1Nrf2TAAMMPSSANAD(P)H:quinone oxidoreductaseEndothelial dysfunctionOrtho-phthalaldehydeOxidative stressCyanidinSODHuman umbilical vein endothelial cellsSuperoxide dismutasecyanidin-3-O-glucosidehypoxia inducible factor-1antioxidant responsive elementNF-E2-related factor-2Total antioxidant activitymatrix metalloproteinaseAREHypoxia
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Authors
S. Anwar, A. Speciale, D. Fratantonio, M. Cristani, A. Saija, F. Virgili, F. Cimino,