| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5874882 | Journal of Stroke and Cerebrovascular Diseases | 2016 | 9 Pages |
Abstract
These findings suggest that EE could promote rehabilitation after ischemia via regulation of p-ERK1/2 expression, which may provide a therapeutic approach for cerebral ischemia or reperfusion injury. The suppression of postischemic astrocytic swelling in the brain of the ischemic rats through the intervention of EE would be one of the underlying mechanisms in the protective effect of cerebral ischemia.
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Authors
Congyu Jiang, Kewei PhD, Yi PhD, Hongyu MD, Gang MD, Junfa PhD, Jie PhD, Shenyi MD,