Article ID Journal Published Year Pages File Type
5879377 The Journal of Pain 2009 11 Pages PDF
Abstract
These findings support the hypothesis that the heightened pain reported by individuals higher in pain catastrophizing may be related to a disruption in the endogenous modulation of pain, operationalized by assessing DNIC. Whether interventions that reduce pain catastrophizing affect pain outcomes via effects on DNIC is in need of investigation.
Related Topics
Life Sciences Neuroscience Neurology
Authors
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