| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5894392 | Placenta | 2015 | 8 Pages |
Abstract
A generalized disruption of genomic imprints leads to triacylglycerol accumulation and abnormal mitochondrial function. This could stem directly from a loss of methylation at a given DMD, such as MEST, or represent a consequence of abnormal placental development.
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Authors
K.P. Himes, A. Young, E. Koppes, D. Stolz, Y. Barak, Y. Sadovsky, J.R. Chaillet,