| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5895149 | Placenta | 2014 | 6 Pages |
Abstract
The increase in NEAT1 and its co-localization with PSPC1 suggests an increase in paraspeckles in IUGR villous trophoblasts. This could lead to an increased retention of important mRNAs in villous trophoblasts nuclei. Given that the villous trophoblasts are crucial for the barrier function of the placenta, this could in part explain placental dysfunction in idiopathic IUGR fetuses.
Keywords
Related Topics
Life Sciences
Biochemistry, Genetics and Molecular Biology
Developmental Biology
Authors
S. Gremlich, F. Damnon, D. Reymondin, O. Braissant, J.C. Schittny, D. Baud, S. Gerber, M. Roth-Kleiner,