| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5902947 | Metabolism | 2016 | 32 Pages |
Abstract
The mechanisms through which regional myocardial function is impaired in MetS and improved consecutive to intervention involved EAT, possibly via paracrine effects of adipocytokines. EAT should be considered as a future therapeutic target of interest in the treatment of metabolic-related cardiac diseases.
Keywords
VVICRPusHOMA-IRTDIHbA1cMyocardial dysfunctionhomeostasis model assessment of insulin resistanceinflammationEpicardial adipose tissueleft ventricle/ventricularCardiovascular diseasesTissue Doppler imagingtumor necrosis factor αEATCVDMetabolic syndromeTNF-αBlood pressureMETSLifestyle interventionStrain ratesGlycated hemoglobinhigh sensitivity C-reactive proteinEpicardial fat
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Authors
Juan Serrano-Ferrer, Edward Crendal, Guillaume Walther, Agnes Vinet, Frédéric Dutheil, Geraldine Naughton, Bruno Lesourd, Robert Chapier, Daniel Courteix, Philippe Obert,