| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5903016 | Metabolism | 2016 | 8 Pages |
Abstract
Insulin sensitivity, inflammation and markers for liver synthetic function differentially impact VLDL-P concentration and VLDL size. These results underscore the complex effects of insulin resistance and its complications on VLDL production.
Keywords
NAFLDVLDLCHSALTIL6nuclear magnetic resonanceaminotransferaseNon-alcoholic steatohepatitisNon-alcoholic steatohepatitis (NASH)interleukin 6Non-alcoholic fatty liver disease (NAFLD)non-alcoholic fatty liver diseaseNMRbody mass indexBMIvery low-density lipoproteinCardiovascular Health StudyInsulin resistanceNash C-reactive proteinCRP
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Authors
Z. Gordon Jiang, Ian H. de Boer, Rachel H. Mackey, Majken K. Jensen, Michelle Lai, Simon C. Robson, Russell Tracy, Lewis H. Kuller, Kenneth J. Mukamal,