| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5943522 | Atherosclerosis | 2016 | 7 Pages |
Abstract
Expression of GALNT3 was reduced in CAD patients, and down regulation of GALNT3 contributed to endothelial injury by promoting apoptosis and up-regulating the expression of MMP-2 and MMP-14 genes via p38 MAPK activation. GALNT3 may be a potential target for future therapeutic intervention for CAD.
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Authors
Liwei Guo, Laiyuan Wang, Hongfan Li, Xueli Yang, Bin Yang, Mengting Li, Jianfeng Huang, Dongfeng Gu,