| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5947422 | Atherosclerosis | 2013 | 10 Pages |
Abstract
The inflammatory status is higher in the immune cells of the carotid plaque, as compared to those cells in the peripheral blood. The altered expression levels of both pro-inflammatory/pro-thrombotic and anti-inflammatory mediators in the milieu of the plaque suggest that the balance between these various mediators may play a key role in carotid disease progression.
Keywords
mTORTLRMNCVCAMPPARJun N-terminal kinaseERKNF-kBPMTMRAATFCDPEGRMCPMMPJnkTNFTGFHMGBCeAMitogen activated proteinMagnetic resonance angiographyCarotid endarterectomyImmunophenotypingPAIplasminogen activator inhibitorGene expressionPeripheral arterial diseaseCerebrovascular diseasecoronary artery diseasetransforming growth factorThrombosisCarotid stenosisToll-like receptorDiabetes mellitusCVDmononuclear cellMononuclear cellsbody mass indexBMICADTissue factortumor necrosis factornuclear factor kappa BBlood pressureAtrial fibrillationcardiovascularmatrix metalloproteinasevascular cell adhesion moleculeheart failurecell surface markersmapmammalian target of rapamycinearly growth responsePADmonocyte chemotactic proteinPeripheralCell adhesionhealthy controlextracellular regulated kinaseperoxisome proliferator-activated receptor
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Zohara Sternberg, Husam Ghanim, Kristen M. Gillotti, Joseph D. Jr., Frederick Munschauer, Richard Curl, Sonya Noor, Jihnhee Yu, Julian L. Sr., Paul Wallace, Paresh Dandona,