Article ID Journal Published Year Pages File Type
5947900 Atherosclerosis 2013 8 Pages PDF
Abstract
A quantitative defect of Tregs, suggestive of decreased peripheral tolerance, could be a potential hallmark of ACS disease. Targeting FOXP3 i l demethylation might elevate the inhibitory activity of Tregs in ACS.
Keywords
Related Topics
Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Preview
FOXP3 demethylation as a means of identifying quantitative defects in regulatory T cells in acute coronary syndrome
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