| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5959975 | Heart Rhythm | 2015 | 27 Pages |
Abstract
Underlying atherosclerosis exacerbates post-MI electrophysiological remodeling and arrhythmias. LPS+MI hearts fully recapitulate the atherosclerotic phenotype, suggesting myocardial inflammation as a key contributor to post-MI arrhythmia.
Keywords
IL-1βN-cadNav1.5apolipoprotein E deficientCD68Connexin43APDOAPN-cadherinMMPLPSCX43AtherosclerosisArrhythmiaMyocardial infarctionApoeapolipoprotein EinflammationelectrocardiogramECGInterleukin-1βleft ventricleVentricular tachycardiaintraperitonealConduction velocityLADlipopolysaccharidematrix metalloproteinaseAction potential durationOptical mappingwild-typevoltage gated sodium channelTransmembrane potentialaction potentialPVCpremature ventricular complexleft anterior descending coronary artery
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Authors
Nicole M. BS, Lianguo MD, Anthony W. BS, Jingjing PhD, Fatemah Shenasa, Donald M. PhD, Merry L. PhD, Crystal M. PhD,