| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5960087 | Heart Rhythm | 2015 | 10 Pages |
Abstract
Enhanced A-V conduction in mice overexpressing SCN5A in the heart mimics the human syndrome of EAVNC. Thus, variants in sodium channel expression in the A-V nodal region may be an electrophysiological substrate responsible for EAVNC.
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Authors
Gong Xin MD, PhD, Carol Ann MD, PhD, Bastiaan J. PhD, Luiz MD, Sridharan PhD,