| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5961134 | Heart Rhythm | 2013 | 25 Pages |
Abstract
These multilevel investigations strongly suggest that Q7R-FGF12 is a disease-associated BrS mutation. Moreover, these data suggest for the first time that FHF effects on Na+ and Ca2+ channels are separable. Most significantly, this study establishes a new method to analyze effects of human arrhythmogenic mutations on cardiac ionic currents.
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Authors
Jessica A. PhD, Cherisse A. PhD, Chuan MD, PhD, Eric Q. BSe, Chaojian PhD, David J. BS, Margherita BcS, Federica MD, Lia MD, PhD, Peter J. MD, PhD, Michael J. MD, PhD, Geoffrey S. MD, PhD,